EBNA-3a–deficient LCLs proliferate extra slowly and display enhanced apoptosis in comparison to wild-kind virus–derived LCLs.
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Yet, peptide pools also can incorporate quite a few shorter peptide sequences that stand for significant immunodominant epitopes of different viruses.
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In addition to that, adoptive T cell therapies like posoleucel also goal LMP2 together with other latency proteins for instance EBNA1, reinforcing its importance as staying a conserved and immunodominant antigen.
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Amid these latency proteins, EBNA-3a performs a supportive but non-essential job in B cell transformation: EBV mutants lacking EBNA-3a can even now renovate B cells, but with substantially diminished performance.
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